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Materials and Methods Cases were 230 patients with incident, histologically confirmed cases of gastric cancer from the Greater Milan area, northern Italy. Controls were 547 frequency-matched subjects admitted to the same network of hospitals as cases for a wide spectrum of acute, non-neoplastic conditions. The DII was computed using a reproducible and valid 78-item food frequency questionnaire. Odds ratios ( OR) were estimated through logistic regression models conditioned on age and sex and adjusted for recognised confounding factors, including total energy intake. How To Install Windows Xp From Usb Flashboot. Introduction Gastric cancer represents the fifth most common cancer and the third-leading cause of cancer death worldwide, with almost 1 million cases and over 700,000 deaths estimated in 2012 (). In Italy, it is the fourth most common cause of cancer death, after lung, colorectal and breast cancers ().

The major recognized risk factor is Helicobacter pylori infection; however, considerable evidence is accumulating on the role of diet and nutrition in the risk of gastric cancer (–). Various dietary components have different effect on inflammation (–). The relation between diet and gastric cancer has been studied widely (, ); however, the possible relation between inflammation deriving from dietary exposure and gastric cancer risk has not yet been investigated. Chronic inflammation is a persistent condition in which tissue destruction and repair occur simultaneously (, ). Evidence from previous studies suggests an important role of chronic inflammation in gastric cancer (–).

A meta-analyses of 5 case-control studies, showed polymorphism of interleukin (IL)-17A G197A to be associated with gastric cancer (). In another meta-analyses, increased pre-treatment serum C-reactive protein level (≥10mg/L) was significantly associated with poor prognosis in gastric cancer patients, either in early or advanced stages (). A literature-derived dietary inflammatory index (DII) was developed to assess the inflammatory potential of an individual’s diet (). The DII has been validated with various inflammatory markers, including C-reactive protein (), interleukin-6 (, ), and homocysteine (). S2k Serials 2011 Download.

The DII has been shown to be associated with metabolic syndrome and it’s components (–), anthropometric measurements and cardiovascular disease in Spain (–); bone mineral density among postmenopausal women in Iran (). Concerning digestive tract cancers, DII is related to an increased risk of colorectal cancer three cohort studies in from the USA (–) and in two cancer case-control studies, in Spain () and Italy (), esophageal squamous cell cancer in three case-control studies (–) and pancreatic cancer in one case-control study(). In addition to cancer incidence DII also was found to be associated with digestive cancer mortality in three cohort studies (–).